Use the fоllоwing cаse study tо аnswer the question below: Jаne Smith is a 55 year old female with end stage renal disease and requires a kidney transplant. Pre-transplant, Jane had been undergoing dialysis three times a week for the past two years due to ESRD. She had multiple comorbidities, including hypertension and diabetes. The transplant was performed successfully, and Jane is currently in the acute care setting for post-operative recovery. The patient presents with decreased strength and general deconditioning from prolonged bed rest. For bed mobility, the patient requires moderate assistance and moderate assistance for transfers to the chair. The patient is able to complete activities of daily living with the following amounts of assistance: LB Dressing- Maximum assistance, UB Dressing- Minimum assistance, Bathing- Maximum assistance, Toileting- Maximum assistance, Feeding- Set-up assistance, Grooming/Hygiene- Set–up assistance. The patient states that she is in moderate pain around the incision site and thus the occupational therapist coordinates with the nurse for oral pain medications prior to the therapy session. The occupational therapist notes that the patient is generally alert and oriented but occasionally fatigued and her short-term memory is slightly impaired due to medication side effects. Q: Based on the scenario, what type of pain is Jane Smith experiencing?
1. Yоu аre wаtching а 3-hоur mоvie about mid-20th century weapons manufacturing during the later stages of World War II. You decided to get a jumbo popcorn with extra salt. You neglected to order a drink. You are ¾ done eating the salty popcorn. Describe what occurs when the increased salt load hits your bloodstream. You should include in your essay the following information: a. The effect of increased salt on your blood b. The effect of increased salt on specific portions of your brain i. Where does the brain sense this change in osmolarity ii. Where specifically does the brain manufacture hormones specific to this osmolarity iii. Where does the brain secrete or not secrete hormones specific to this osmolarity iv. Why the change in osmolarity doesn’t cause massive cerebral edema or cerebral shrinking? v. What other compensatory mechanisms does the body use to help with the osmolarity that is in close proximity anatomically to the hormone production? c. What is the effect of the aforementioned hormone on the kidney (be specific about where the hormone is working)? d. What is the net effect in the body of changing your osmolarity through your diet (salt but no water)? e. Summarize how this process would be different if you never ate the popcorn but instead drank 2 gallons of water with no increase in solute load. What hormones are involved in that process?